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Under HO-induced oxidative stress, the expression levels of -catenin and its downstream effector CDH3 are markedly reduced in melanocytes, thereby weakening their antioxidant responses [135].Vitamin D treatment reverses this suppression by promoting Ser9 phosphorylation of GSK3, thereby preventing -catenin degradation and activating downstream transcriptional programs

During the same period, estrone (E1) becomes the main circulating estrogen, but its activity is only one-tenth that of E2

The future holds great potential for personalized, multi-targeted therapies, innovative delivery systems, and a more comprehensive understanding of TBI, ultimately leading to improved outcomes and quality of life for TBI patients