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Furthermore, the early consideration that NO produced by activated macrophages would inactivate the iron/sulfur centers in tumor cell mitochondria (329, 542) has been reevaluated by a series of experiments indicating that the NO-dependent inactivation of iron/sulfur centers was in fact mediated by peroxynitrite (182, 523)

4.2 New dimensions of barrier disruption: ferroptosis propagation and mitochondrial metabolic-immune crosstalk With the deepening understanding of the pathological mechanisms of sepsis-associated ARDS, research hotspots are gradually shifting from classical apoptosis to more destructive forms of regulated cell death (44)