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IR induced metabolic disturbances lead to degradation of brain function, and prolonged uncontrolled high blood glucose levels adversely affect a variety of metabolic pathways such as oxidative stress, formation of advanced glycosylation end products (AGEs), and protein kinase C activation, while metabolic disturbances inducing cerebral microvascular damage affect the extent of cognitive impairment (1, 7)

P53 mutation suppresses adult neurogenesis in medaka fish (oryzias latipes)

We have previously shown that mitogen-activated protein kinase (MAPK) and phosphatidylinositol 3-kinase (PI3K) are the major downstream signalling molecules that mediate HGF-induced proliferation and migration of cancer and endothelial cells

In their study, it was suggested that GHK-Cu possesses protective effects in lipopolysaccharide-induced acute lung injury