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Therefore, the model provides a platform from which the impact of varying degrees of quinone concentration and exposure times on GSH homeostasis may be computationally examined

Although these results highlight a role for KU-57788-activated DRP1 in modulating the NRF2/SLC7A11/GSH axis, we cannot exclude the possibility that KU-57788 exerts its anti-tumor effects through additional parallel mechanisms

6 Growth factors, PRP, and biologics Growth factors, such as TGF- (Sun et al., 2023

The identification of GAPDH as an innate NET-suppressor mechanism suggests that preservation of GAPDH activity may be important to prevent NET formation in hyperinflammatory conditions characterized by oxidative stress