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Grafts subjected to ischemia are more vulnerable to fungal infections due to their compromised blood flow, which restricts the delivery of oxygen and nutrients to the site of infection

This suggests extracellular metabolism happens before cellular uptake [18]

Rather, the defect in heme synthesis was explained by an inhibition of ferrochelatase activity by a still ill-defined mechanism as well as an increased level of zinc leading to the accumulation of Zn-PPIX (Schoenfeld et al., 2005)