glutathione s-transferase gsts catalyze the conjugation Functional and Structural Diversity of Insect S-transferases in Xenobiotic Adaptation Mechanism of the glutathione conjugation

Excessive pro-inflammatory response activates ATF-3 and inducible nitric oxide synthase (iNOS), which leads to the occurrence of oxidative stress and becomes another cause of prolonged wound healing

The tumor suppressor miR-9 bound to the mRNA of human epidermal growth factor 2 (HER2) and increased the response to cyclophosphamide [49]

For xenograft studies, animals were allocated into groups ensuring the mean, median and standard error of tumor size was similar across all groups

They help regulate the balance between reduced glutathione (GSH) and oxidized glutathione (GSSG), which is crucial for normal cellular processes and signaling